Search results for "Heart malformation"

showing 9 items of 9 documents

An overview of the molecular mechanisms underlying development and progression of bicuspid aortic valve disease

2019

Bicuspid aortic valve (BAV) is a common congenital heart malformation frequently associated with the development of aortic valve diseases and severe aortopathy, such as aortic dilatation, aneurysm and dissection. To date, different genetic loci have been identified in syndromic and non- syndromic forms of BAV. Among these, genes involved in the regulation of extracellular matrix remodelling, epithelial to mesenchymal transition and nitric oxide metabolism appear to be the main contributors to BAV pathogenesis. However, no- single gene model explains BAV inheritance, suggesting that more factors are simultaneously involved. In this regard, characteristic epigenetic and immunological profiles…

0301 basic medicineaneurysm; aortic dilatation; aortic stenosis; aortopathy; bicuspid aortic valve; NOTCH1Aortic stenosibicuspid aortic valveHeart malformationAortic DiseasesHeart Valve Diseasesaortopathy610 Medicine & healthDisease030204 cardiovascular system & hematologyBioinformatics2705 Cardiology and Cardiovascular Medicine11459 Center for Molecular CardiologyPathogenesis03 medical and health sciences0302 clinical medicineAneurysmBicuspid aortic valveNOTCH1Bicuspid Aortic Valve Disease1312 Molecular BiologymedicineSettore MED/05 - Patologia ClinicaAnimalsHumansEpithelial–mesenchymal transitionEpigeneticsMolecular BiologyAortic dilatationbusiness.industryaortic stenosisaortic dilatationmedicine.disease030104 developmental biologyAortic ValveaneurysmDisease Progressioncardiovascular systemCardiology and Cardiovascular MedicinebusinessJournal of Molecular and Cellular Cardiology
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In Vivo Cardiotoxicity Induced by Sodium Aescinate in Zebrafish Larvae

2016

Sodium aescinate (SA) is a widely-applied triterpene saponin product derived from horse chestnut seeds, possessing vasoactive and organ-protective activities with oral or injection administration in the clinic. To date, no toxicity or adverse events in SA have been reported, by using routine models (in vivo or in vitro), which are insufficient to predict all aspects of its pharmacological and toxicological actions. In this study, taking advantage of transparent zebrafish larvae (Danio rerio), we evaluated cardiovascular toxicity of SA at doses of 1/10 MNLC, 1/3 MNLC, MNLC and LC10 by yolk sac microinjection. The qualitative and quantitative cardiotoxicity in zebrafish was assessed at 48 h p…

0301 basic medicinesodium aescinateEmbryo NonmammalianHeart malformationDrug Evaluation PreclinicalPharmaceutical Science010501 environmental sciencesPharmacology01 natural sciencesAnalytical ChemistryHeart RateDrug DiscoveryToxicity Tests ChronicZebrafishYolk SacbiologyCommunicationHeartLC10medicine.anatomical_structureChemistry (miscellaneous)LarvaToxicityMolecular MedicineHeart Defects CongenitalMicroinjectionscardiotoxicityHemorrhagelarvaelcsh:QD241-44103 medical and health scienceslcsh:Organic chemistryIn vivoHeart ratemedicineMNLCAnimalsPhysical and Theoretical ChemistryYolk sacAdverse effect0105 earth and related environmental sciencesCardiotoxicityDose-Response Relationship DrugOrganic ChemistryThrombosisSaponinsbiology.organism_classificationzebrafishTriterpenes030104 developmental biologyMolecules
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Hypoplasia of the posterior mitral valve leaflet: Don't forget to look beyond the mitral valve.

2020

Hypoplasia of the posterior mitral valve leaflet (PMVL) is a very rare finding in adulthood and can coexist with other congenital heart defects. In this image, a transesophageal echocardiography (TOE) carried out on a 59-year-old woman with a 2-month history of dyspnea revealed a hypoplastic PMVL causing severe mitral regurgitation associated with a secundum-type atrial septal defect (ASD) with left-to-right shunting. This case demonstrates how essential 3-dimensional TOE is for a comprehensive assessment of the mitral valve and to improve the diagnostic accuracy of concomitant congenital heart abnormalities.

Adultmedicine.medical_specialtyHeart malformationDiagnostic accuracy030204 cardiovascular system & hematologyHeart Septal Defects AtrialPosterior mitral valve leaflet03 medical and health sciences0302 clinical medicineMitral valveInternal medicinemedicineHumansRadiology Nuclear Medicine and imagingcardiovascular diseases030212 general & internal medicineMitral regurgitationMitral Valve Prolapsebusiness.industryMitral Valve InsufficiencyMiddle Agedmedicine.diseaseHypoplasiaShuntingmedicine.anatomical_structurecardiovascular systemCardiologyMitral ValveFemaleCardiology and Cardiovascular MedicinebusinessEchocardiography TransesophagealEchocardiography (Mount Kisco, N.Y.)REFERENCES
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Missense variants in TAF1 and developmental phenotypes: Challenges of determining pathogenicity

2019

We recently described a new neurodevelopmental syndrome (TAF1/MRXS33 intellectual disability syndrome) (MIM# 300966) caused by pathogenic variants involving the X-linked gene TAF1, which participates in RNA polymerase II transcription. The initial study reported eleven families, and the syndrome was defined as presenting early in life with hypotonia, facial dysmorphia, and developmental delay that evolved into intellectual disability (ID) and/or autism spectrum disorder (ASD). We have now identified an additional 27 families through a genotype-first approach. Familial segregation analysis, clinical phenotyping, and bioinformatics were capitalized on to assess potential variant pathogenicity…

Genetics0303 health sciencesHeart malformation030305 genetics & heredityBiologymedicine.diseaseArticleHypotonia03 medical and health sciencesAutism spectrum disorderHuman Phenotype OntologyIntellectual disabilityGeneticsmedicineCopy-number variationAllelemedicine.symptomGenetics (clinical)Exome sequencing030304 developmental biologyHuman Mutation
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Redefining the MED13L syndrome

2015

Congenital cardiac and neurodevelopmental deficits have been recently linked to the mediator complex subunit 13-like protein MED13L, a subunit of the CDK8-associated mediator complex that functions in transcriptional regulation through DNA-binding transcription factors and RNA polymerase II. Heterozygous MED13L variants cause transposition of the great arteries and intellectual disability (ID). Here, we report eight patients with predominantly novel MED13L variants who lack such complex congenital heart malformations. Rather, they depict a syndromic form of ID characterized by facial dysmorphism, ID, speech impairment, motor developmental delay with muscular hypotonia and behavioral difficu…

MaleAdolescentHeart malformationTransposition of Great VesselsRNA polymerase IIBioinformaticsArticleMediatorIntellectual DisabilityIntellectual disabilityGeneticsmedicineTranscriptional regulationHumansAbnormalities MultipleChildTranscription factorGenetics (clinical)GeneticsScience & TechnologyMediator ComplexbiologyMuscular hypotoniaSyndromemedicine.diseasePhenotypeChild PreschoolMutationbiology.proteinMuscle HypotoniaFemaleNeurocognitiveEuropean Journal of Human Genetics
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LARP7 variants and further delineation of the Alazami syndrome phenotypic spectrum among primordial dwarfisms: 2 sisters.

2019

Abstract Alazami syndrome (AS) (MIM# 615071 ) is an autosomal recessive microcephalic primordial dwarfism (PD) with recognizable facial features and severe intellectual disability due to depletion or loss of function variants in LARP7. To date, 15 patients with AS have been reported. Here we describe two consanguineous Algerian sisters with Alazami PD due to LARP7 homozygous pathogenic variants detected by whole exome sequencing. By comparing these two additional cases with those previously reported, we strengthen the key features of AS: severe growth restriction, severe intellectual disability and some distinguishing facial features such as broad nose, malar hypoplasia, wide mouth, full li…

medicine.medical_specialtyHeart malformation[SDV]Life Sciences [q-bio]Dwarfism03 medical and health sciencesLoss of Function MutationIntellectual DisabilityIntellectual disabilityGeneticsmedicineHumansChildGenetics (clinical)Exome sequencingLoss function030304 developmental biology0303 health sciencesbusiness.industrySiblings030305 genetics & heredityGeneral MedicineSyndromemedicine.diseaseDermatologyPhenotype[SDV] Life Sciences [q-bio]PhenotypeRibonucleoproteinsEtiologyMicrocephalyFemalePrimordial dwarfismbusinessMild microcephalyEuropean journal of medical genetics
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Are Kawasaki Disease and Pediatric Multi-Inflammatory Syndrome Two Distinct Entities? Results from a Multicenter Survey During SARS-CoV-2 Epidemic in…

2020

Background: There is mounting evidence on the existence of a childhood multi-inflammatory syndrome related to SARS-CoV-2, sharing similarities with Kawasaki Disease (KD).  Methods: On April 24th,2020 the Rheumatology Study Group of the Italian Pediatric Society launched a survey to enroll patients diagnosed with KD or KD-like disease. Classification was: 1) classical and incomplete KD, named Kawasaki Disease Group (KDG); 2) KD-like multi-inflammatory syndrome, named KawaCOVID Group (KCG). Demographic, clinical, and laboratory data, treatment information, and patients’ outcome were collected in an online anonymized database (RedCAPO). Chi square test or exact Fisher test and non parametric W…

medicine.medical_specialtyMyocarditisHeart malformationbusiness.industryDiseasemedicine.diseaseInstitutional review boardsymbols.namesakeInternal medicineCohortChi-square testsymbolsMedicineKawasaki diseasebusinessFisher's exact testSSRN Electronic Journal
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An Exploratory Look at Bicuspid Aortic Valve (Bav) Aortopathy: Focus on Molecular and Cellular Mechanisms

2019

Bicuspid aortic valve (BAV) is the most common congenital heart malformation. BAV patients are at increased risk for aortic valve disease (stenosis/regurgitation), infective endocarditis, thrombi formation and, in particular, aortic dilatation, aneurysm and dissection. This review aims at exploring the possible interplay among genetics, extracellular matrix remodeling, abnormal signaling pathways, oxidative stress and inflammation in contributing to BAV-associated aortopathy (BAV-A-A). Novel circulating biomarkers have been proposed as diagnostic tools able to improve risk stratification in BAV-A-A. However, to date, the precise molecular and cellular mechanisms that lead to BAV-A-A remain …

medicine.medical_specialtySettore MED/09 - Medicina InternaBicuspid aortic valveHeart malformationBicuspid aortic valve; atherosclerosisInflammationRegurgitation (circulation)030204 cardiovascular system & hematologymedicine.disease_cause03 medical and health sciences0302 clinical medicineBicuspid aortic valveAneurysmBicuspid Aortic Valve DiseaseInternal medicinemedicineHumans030212 general & internal medicinebusiness.industryHemodynamicsGeneral Medicinemedicine.diseaseVulnerable plaqueStenosisInfective endocarditisAortic ValveCardiologymedicine.symptomatherosclerosisbusinessCardiology and Cardiovascular MedicineBiomarkers
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Congenitally corrected transposition of great vessels: MRI and echocardiographic appearance

1995

This paper presents the case of a 36-year-old male with congenitally corrected transposition of great vessels (CTGV). This is a rare cardiac anomaly characterized by atrioventricular and ventriculoarterial discordance, occuriing in less than 1% of all congenital heart diseases. This was diagnosed by echocardiography and MRI. The MRI findings demonstrated cardiac abnormality.

medicine.medical_specialtymedicine.diagnostic_testbusiness.industryHeart malformationUltrasoundInterventional radiologyGeneral MedicineCongenitally corrected transpositionGreat vesselsInternal medicinecardiovascular systemmedicineCardiologyCARDIAC ANOMALYRadiology Nuclear Medicine and imagingRadiologybusinessMri findingsNeuroradiologyEuropean Radiology
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